Understanding Of APA Formatting Conventions

Develop: Understanding of APA Formatting Conventions
Evaluation Title: APA Formatting: You’re the Instructor
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McStudent 1

Information Literacy Skills in the College Classroom

Student McStudent

Herzing University

EN 111: Information Literacy

Professor Smith

April 27, 2020

McStudent 2

Introduction

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McStudent 3

BIBLIOGRAPHY

Hanson, L. F. (2018). Fundamentals of information literacy. Journal of Library Studies, 55(11), 893-896.

http://doi.org/gh6ty8

Alvarez, Juan, and Julio A. Camargo. (2016, Jan). “Undergraduate Perception of Information Literacy:

Defining, Attaining and Self-Assessing Skills.” College & Research Libraries, vol. 21, no. 1, pp. 90-

94. https://doi.org/11.1342/rev0000128
Explain each error in a numbered list, using full sentences for each.

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Your assignment submission should be a Word document that fully adheres to the instructions listed above. Be sure to proofread your assignment.

 
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Human Pathophysiology.

HCR 240 Module 1 Learning Guide

pg. 1 of 4

MODULE REFLECTION – In the box below, reflect on the course material and activities for this module. If you did not complete all the activities, did you have any questions or comments relating to this? What content did you find the most interesting and why? This should be a 4-5 sentence paragraph.

 

 

 

 

MODULE CONTENT REVIEW STUDY GUIDE CHAPTER 2 – CELLULAR INJURY, ADAPTATIONS, AND MALADAPTIVE CHANGES Define the following terms:

Term Definition

Etiology

Histology

Biopsy

Pathognomic Changes

 

Atrophy

Hypertrophy

Hyperplasia

Metaplasia

Dysplasia

Neoplasia

Hypoxia

Apoptosis

Necrosis

Infarction

Gangrene

 

 

HCR 240 Module 1 Learning Guide

pg. 2 of 4

 

CHAPTER 3 – GENETIC BASIS OF DISEASE Genetic Diseases to Know – Fill in the table below with information from textbook or videos regarding type of genetic or chromosomal disorder occurs, signs and symptoms, diagnostic testing, and possible treatments:

Genetic Disease or Syndrome

Type of Genetic / Chromosomal Disorder

Signs and Symptoms of Disease

Diagnostic Testing Possible Treatments

Cystic Fibrosis

 

Tay Sachs Disease

 

Down Syndrome

 

Huntington’s Disease

 

CHAPTER 9 – INFLAMMATION AND DYSFUNCTIONAL WOUND HEALING Define and give examples of the terms below using your textbook and/or videos:

Term Definition Examples

Acute inflammation

Purulent exudate

Abscess

Transudate

Effusion

Leukocytosis

Chemotaxis

 

 

HCR 240 Module 1 Learning Guide

pg. 3 of 4

Term Definition Examples

Cytokines

Fibrinogen

White Blood Cells

Pyrogens

Lymphadenopathy

Histamine

Chronic inflammation

Four phases of wound healing

 

Hemostasis

Wound retraction

Primary intention

Secondary intention

Tertiary intention

Eschar

Debridement

Wound dehiscence

Wound evisceration

Keloid

Contractures

Stricture

Fistula

Adhesion

 

 

 

 

 

HCR 240 Module 1 Learning Guide

pg. 4 of 4

CHAPTER 10 – INFECTIOUS DISEASES These are the required infectious diseases you should know. Fill in the table to answer type, signs, and symptoms of each:

Pathogen Type (Bacteria, Virus, Fungus, or Parasite)

Signs and Symptoms One Interesting Fact / Statistic of Disease

Staphylococcal Infections

 

Streptococcal Infections

 

Pneumonia Infections

 

Bacterial Meningitis: Haemophiles influenzae

 

GI Tract Infections – Escherichia coli

 

Common Cold

Influenza

Epstein Barr

MMR – Measles, Mumps, Rubella

 

Varicella

Herpes Simplex

Candida

Malaria

 

 

  • Chapter 2 – Cellular Injury, Adaptations, and Maladaptive Changes
  1. DefinitionEtiology: original cause of cell alteration/disease
  2. DefinitionHistology: microscopic study of cells/tissues
  3. DefinitionBiopsy: sample of histological analysis
  4. DefinitionPathognomic Changes: histological findings that represent disease
  5. DefinitionAtrophy: cells shrink in size due to reduction of metabolic demand
  6. DefinitionHypertrophy: increase in cell size
  7. DefinitionHyperplasia: increase number of cells to organ/tissue
  8. DefinitionMetaplasia: replace one cell type with another
  9. DefinitionDysplasia: derranged cellular growth, result of chronic inflammation, or precancerous condition
  10. DefinitionNeoplasia: “new growth” known to be disorganized, uncoordinated, cancerous.
  11. DefinitionHypoxia: diminished O2 to cells, due to ischemia, problem’s with RBC’s, and pulmonary issues
  12. DefinitionApoptosis: programmed cell death. does not cause inflammation
  13. DefinitionNecrosis: cell death due to injury-initiates inflammatory reaction
  14. DefinitionInfarction: ischemic necrosis
  15. DefinitionGangrene: prolonged ischemia, infarction, and necrosis
  16. Type of Genetic Chromosomal DisorderCystic Fibrosis:
  17. Signs and Symptoms of DiseaseCystic Fibrosis:
  18. Diagnostic TestingCystic Fibrosis:
  19. Possible TreatmentsCystic Fibrosis:
  20. Type of Genetic Chromosomal DisorderTay Sachs Disease:
  21. Signs and Symptoms of DiseaseTay Sachs Disease:
  22. Diagnostic TestingTay Sachs Disease:
  23. Possible TreatmentsTay Sachs Disease:
  24. Type of Genetic Chromosomal DisorderDown Syndrome:
  25. Signs and Symptoms of DiseaseDown Syndrome:
  26. Diagnostic TestingDown Syndrome:
  27. Possible TreatmentsDown Syndrome:
  28. Type of Genetic Chromosomal DisorderHuntingtons Disease:
  29. Signs and Symptoms of DiseaseHuntingtons Disease:
  30. Diagnostic TestingHuntingtons Disease:
  31. Possible TreatmentsHuntingtons Disease:
  32. DefinitionAcute inflammation:
  33. ExamplesAcute inflammation:
  34. DefinitionPurulent exudate:
  35. ExamplesPurulent exudate:
  36. DefinitionAbscess:
  37. ExamplesAbscess:
  38. DefinitionTransudate:
  39. ExamplesTransudate:
  40. DefinitionEffusion:
  41. ExamplesEffusion:
  42. DefinitionLeukocytosis:
  43. ExamplesLeukocytosis:
  44. DefinitionChemotaxis:
  45. ExamplesChemotaxis:
  46. DefinitionCytokines:
  47. ExamplesCytokines:
  48. DefinitionFibrinogen:
  49. ExamplesFibrinogen:
  50. DefinitionWhite Blood Cells:
  51. ExamplesWhite Blood Cells:
  52. DefinitionPyrogens:
  53. ExamplesPyrogens:
  54. DefinitionLymphadenopathy:
  55. ExamplesLymphadenopathy:
  56. DefinitionHistamine:
  57. ExamplesHistamine:
  58. DefinitionChronic inflammation:
  59. ExamplesChronic inflammation:
  60. DefinitionFour phases of wound healing:
  61. ExamplesFour phases of wound healing:
  62. DefinitionHemostasis:
  63. ExamplesHemostasis:
  64. DefinitionWound retraction:
  65. ExamplesWound retraction:
  66. DefinitionPrimary intention:
  67. ExamplesPrimary intention:
  68. DefinitionSecondary intention:
  69. ExamplesSecondary intention:
  70. DefinitionTertiary intention:
  71. ExamplesTertiary intention:
  72. DefinitionEschar:
  73. ExamplesEschar:
  74. DefinitionDebridement:
  75. ExamplesDebridement:
  76. DefinitionWound dehiscence:
  77. ExamplesWound dehiscence:
  78. DefinitionWound evisceration:
  79. ExamplesWound evisceration:
  80. DefinitionKeloid:
  81. ExamplesKeloid:
  82. DefinitionContractures:
  83. ExamplesContractures:
  84. DefinitionStricture:
  85. ExamplesStricture:
  86. DefinitionFistula:
  87. ExamplesFistula:
  88. DefinitionAdhesion:
  89. ExamplesAdhesion:
  90. Type Bacteria Virus Fungus or ParasiteStaphylococcal Infections:
  91. Signs and SymptomsStaphylococcal Infections:
  92. One Interesting Fact Statistic of DiseaseStaphylococcal Infections:
  93. Type Bacteria Virus Fungus or ParasiteStreptococcal Infections:
  94. Signs and SymptomsStreptococcal Infections:
  95. One Interesting Fact Statistic of DiseaseStreptococcal Infections:
  96. Type Bacteria Virus Fungus or ParasitePneumonia Infections:
  97. Signs and SymptomsPneumonia Infections:
  98. One Interesting Fact Statistic of DiseasePneumonia Infections:
  99. Type Bacteria Virus Fungus or ParasiteBacterial Meningitis Haemophiles influenzae:
  100. Signs and SymptomsBacterial Meningitis Haemophiles influenzae:
  101. One Interesting Fact Statistic of DiseaseBacterial Meningitis Haemophiles influenzae:
  102. Type Bacteria Virus Fungus or ParasiteGI Tract Infections Escherichia coli:
  103. Signs and SymptomsGI Tract Infections Escherichia coli:
  104. One Interesting Fact Statistic of DiseaseGI Tract Infections Escherichia coli:
  105. Type Bacteria Virus Fungus or ParasiteCommon Cold:
  106. Signs and SymptomsCommon Cold:
  107. One Interesting Fact Statistic of DiseaseCommon Cold:
  108. Type Bacteria Virus Fungus or ParasiteInfluenza:
  109. Signs and SymptomsInfluenza:
  110. One Interesting Fact Statistic of DiseaseInfluenza:
  111. Type Bacteria Virus Fungus or ParasiteEpstein Barr:
  112. Signs and SymptomsEpstein Barr:
  113. One Interesting Fact Statistic of DiseaseEpstein Barr:
  114. Type Bacteria Virus Fungus or ParasiteMMR Measles Mumps Rubella:
  115. Signs and SymptomsMMR Measles Mumps Rubella:
  116. One Interesting Fact Statistic of DiseaseMMR Measles Mumps Rubella:
  117. Type Bacteria Virus Fungus or ParasiteVaricella:
  118. Signs and SymptomsVaricella:
  119. One Interesting Fact Statistic of DiseaseVaricella:
  120. Type Bacteria Virus Fungus or ParasiteHerpes Simplex:
  121. Signs and SymptomsHerpes Simplex:
  122. One Interesting Fact Statistic of DiseaseHerpes Simplex:
  123. Type Bacteria Virus Fungus or ParasiteCandida:
  124. Signs and SymptomsCandida:
  125. One Interesting Fact Statistic of DiseaseCandida:
  126. Type Bacteria Virus Fungus or ParasiteMalaria:
  127. Signs and SymptomsMalaria:
  128. One Interesting Fact Statistic of DiseaseMalaria:
  129. ModuleReflection:
 
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Neurological And Musculoskeletal Disorders

Patient is a 24-year-old female administrative assistant who comes to the emergency department with a chief complaint of severe right-sided headache. She states that this is the sixth time in the last 2 months she has had this headache. She says the headaches last 2–3 days and have impacted her ability to concentrate at work. She complains of nausea and has vomited three times in the last 3 hours. She states, “the light hurts my eyes.” She rates her pain as a 10/10 at this time. Ibuprofen and acetaminophen ease her symptoms somewhat but do not totally relieve them. No other current complaints.

Assignment (2- to 3-page case study analysis)

In the Case Study Analysis related to the scenario provided, explain the following:

  • Both the neurological and musculoskeletal pathophysiologic processes that would account for the patient presenting these symptoms.
  • Any      racial/ethnic variables that may impact physiological functioning.
  • How      these processes interact to affect the patient.
 
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Physiology Case Analysis

Teri goes to see her PCP after feeling depressed last couple of months as well as experiencing a seizure earlier this week. The physician orders a few test, results are shown below:

  • Low hematocrit
  • Low levels of Vit B12
  • Low Oxygen levels
  • Presence of autoantibodies against the parietal cell
  • Peripheral blood smear shows abnormal RBC (large pale RBCs)

After viewing the results, Teri is diagnosed with Pernicious Anemia; a condition in which autoantibodies destroys the parietal cells found within the stomach.

Address each of the following regarding Teri’s case:

  1. Since the parietal cell is destroyed, which substance(s) is Teri lacking as a consequence?
  2. What is the correlation between destruction of parietal cells and Vit B12 deficiency?
  3. Due to her condition, Teri lacks Pepsin. Explain why.
  4. If Teri were healthy, explain what her RBCs would look like?
  5. What is hematocrit?
  6. Low oxygen levels stimulate her body to create new RBCs by stimulating which process? Explain that process in depth.
  7. What protein transports oxygen in our body and explain its structure?
  8. What cell secretes antibodies and explain the various functions of antibodies.
  9. What are the different types of antibodies and give two characteristics for each?
  10. Explain the structure of an immunoglobulin.

Specifics:

  • Support your opinions with evidence from your readings and research.
  • You must reference a minimum of three (3) scholarly sources in your paper, which includes one non-Internet source.
  • Use current APA Style to format your paper and to cite your sources.
  • Your paper/total responses must be 3– to 4-pages in length not including the title page, abstract, and reference pages.
    • Title page: Case Title, Full Name, Section, Date, Instructor and Campus
    • Abstract: Summarize the case in a paragraph
    • Introduction: The physiological importance of the digestive system, immune system, and cardiovascular system for the proper functioning of the human body
    • Body: Organize your analysis with headings that thoroughly answer the prompts
    • Conclusion: Summarize the paper in a paragraph Teri goes to see her PCP after feeling depressed last couple of months as well as experiencing a seizure earlier this week. The physician orders a few test, results are shown below:
      • Low hematocrit
      • Low levels of Vit B12
      • Low Oxygen levels
      • Presence of autoantibodies against the parietal cell
      • Peripheral blood smear shows abnormal RBC (large pale RBCs)

      After viewing the results, Teri is diagnosed with Pernicious Anemia; a condition in which autoantibodies destroys the parietal cells found within the stomach.

      Address each of the following regarding Teri’s case:

      1. Since the parietal cell is destroyed, which substance(s) is Teri lacking as a consequence?
      2. What is the correlation between destruction of parietal cells and Vit B12 deficiency?
      3. Due to her condition, Teri lacks Pepsin. Explain why.
      4. If Teri were healthy, explain what her RBCs would look like?
      5. What is hematocrit?
      6. Low oxygen levels stimulate her body to create new RBCs by stimulating which process? Explain that process in depth.
      7. What protein transports oxygen in our body and explain its structure?
      8. What cell secretes antibodies and explain the various functions of antibodies.
      9. What are the different types of antibodies and give two characteristics for each?
      10. Explain the structure of an immunoglobulin.

      Specifics:

      • Support your opinions with evidence from your readings and research.
      • You must reference a minimum of three (3) scholarly sources in your paper, which includes one non-Internet source.
      • Use current APA Style to format your paper and to cite your sources.
      • Your paper/total responses must be 3– to 4-pages in length not including the title page, abstract, and reference pages.
        • Title page: Case Title, Full Name, Section, Date, Instructor and Campus
        • Abstract: Summarize the case in a paragraph
        • Introduction: The physiological importance of the digestive system, immune system, and cardiovascular system for the proper functioning of the human body
        • Body: Organize your analysis with headings that thoroughly answer the prompts
        • Conclusion: Summarize the paper in a paragraph
 
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